Unraveling Autistic Consciousness
A Phenomenological Perspective on Perception, Memory, and the Bergsonian Mind
Palavras-chave:
Autism, Bergsonian Philosophy, Motor Stereotypies, Phenomenology and Cognitive SchemaResumo
This article explores the underlying mechanisms of motor and language disorders in autism, employing a propositional critique based on Henri Bergson's philosophical framework. Building upon Eugène Minkowski's application of Bergsonian philosophy, this study reexamines theoretical assumptions in psychiatric research, with a particular focus on schizophrenia and its parallels to autism. In the field of phenomenological studies, this investigation pioneers the application of Bergsonian analysis to autism, dissecting non-social behaviors like stereotypies, echolalia, and exceptional memory. More specifically, based on these propositions, we challenge the prevailing assumption of a dichotomy between physical and subjective states that underpins autism research in psychiatry and cognitive psychology. On the contrary, we propose that autism signifies a comprehensive alteration in existential experience, rather than a collection of isolated symptoms. Therefore, establishing as a fundamental idea the existence of an imbalance between perception and memory, we consider that speech and motor "stereotypes", more than mere repetitive and involuntary acts, are part of the subject's creative effort to integrate self-experience. (In regard to) Concerning the conception of cognition, we argue for a broader cognitive model in autism that integrates brain function with motor schemas and environmental interactions. In fact, by examining the problem in its historical, philosophical, and cognitive dimensions, our analysis endeavors to enrich dialogues on pathological phenomenology and consciousness, while critically appraising modern interpretations of autism that overemphasize organic causality. Ultimately, we question the reductionist view that attributes autistic behaviors solely to organic causes and prioritizes social interaction deficits.